Macroangiopathy mob ntshav qab zib mellitus - cov laj thawj thiab kev kho mob

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Ntshav qab zib Macroangiopathy - lub sij hawm ua ke los ntawm qhov twg atherosclerosis ntawm cov hlab ntsha loj yog to taub. Mob ntshav qab zib ua rau kev loj hlob ntawm tus kabmob, uas yog nrog cov ntshav nce ntawm lub cev. Hauv qhov no, cov txheej txheem metabolic, nrog rau cov rog hauv cov metabolism, cuam tshuam. Qhov no ua rau kev tsim cov atherosclerotic plaques ntawm cov leeg ntsa. Ua ntej tshaj plaws, lub siab, lub hlwb thiab txhais ceg raug kev txom nyem.

Vim li cas

Ntau yam ua rau kev txhim kho ntawm tus kab mob no:

  • Lub cev hnyav dhau;
  • Cov cwj pwm tsis zoo - haus cawv thiab haus luam yeeb;
  • Tawg siab
  • Kev tsim kho ntawm atrial fibrillation;
  • Nce cov roj cholesterol;
  • Hnub nyoog tshaj 50 xyoo;
  • Kev tshuaj ntsuam genetic predisposition.

Ntxiv rau, muaj qee yam qhia txog kev mob ntshav qab zib. Cov laj thawj no suav nrog cov hauv qab no:

  • Hyperglycemia;
  • Nce insulin ntau ntau - qhov mob no yog hu ua hyperinsulinemia;
  • Kev tiv thaiv kab mob rau kev cuam tshuam ntawm lub cev lawm - tus mob no yog hu ua insulin tsis kam;
  • Kab mob rau raum nrog ntshav qab zib;
  • Lub sijhawm ntev ntawm tus kabmob.

Ib qhov ua rau tseem ceeb ntawm atherosclerotic cov txheej txheem hauv ntshav qab zib yog qhov kev tso tawm ntawm cov insulin ntau hauv cov ntshav. Cov kab mob no tuaj yeem ua tus kab mob ischemic.

Insulin provokes cov tsos mob ntawm plaques cov roj ntsha thiab cov lipoprotein tawg. Qhov no tuaj yeem yog qhov tshwm sim ncaj qha rau cov phab ntsa ntsa los yog cov nyhuv ntawm lipid metabolism.

Kev faib tawm thiab kuaj mob nthuav tawm

Ntshav qab zib macroangiopathy tuaj yeem muaj ntau txoj hauv kev loj hlob. Txhua daim ntawv ntawm pathology yog tsiag ntawv los ntawm tej yam ntxwv.

Nrog kev puas tsuaj rau cov hlab ntsha, qhov tshwm sim ntawm angina pectoris yog cai. Qhov kev ua txhaum no yog txuam nrog kev ua txhaum ntawm cov txheej txheem kev pabcuam ntshav. Nws ua tau nws tus kheej hauv daim ntawv ntawm kev mob siab hauv pob tw. Kuj tseem muaj kev pheej hmoo ntawm kev tsim myocardial infarction thiab mob plawv nres.

Daim ntawv ntawm pathology yog tsiag ntawv los ntawm cov kev qhia:

  1. Kev nias, hlawv, nyem qhov mob ntawm ib cheeb tsam ntawm lub siab thiab hauv pob tw. Nyob rau theem pib ntawm kev txhim kho tus kabmob, lawv tshwm sim nkaus xwb nrog kev tawm dag zog lub cev. Raws li lawv txhim kho, tsis xis nyob yog tam sim no nyob rau hauv lub xeev siab txawm tias tom qab siv tshuaj los ntawm qeb ntawm nitrates.
  2. Ua tsis taus pa. Thaum xub thawj, nws raug pom tsuas yog nyob rau hauv lub nra hnyav, thiab tom qab ntawd ces nyob hauv lub xeev khov kho.
  3. O o ntawm cov ceg.
  4. Kev ua haujlwm tsis zoo ntawm lub siab.
  5. Ua rau cov ntshav siab ntxiv.
  6. Tsis mob plawv. Qhov kab ke no feem ntau pom nyob rau hauv cov ntshav qab zib. Qhov no yog vim muaj qhov ua tsis zoo ntawm cov hlab ntaws.

Kev puas tsuaj rau cov hlab ntsha ntawm lub hlwb yog hu ua cerebrovascular pathology. Nrog nws txoj kev loj hlob, cov kev qhia pom muaj:

  1. Mob taub Hau.
  2. Deterioration ntawm kev tshaj tawm.
  3. Kiv taub hau
  4. Lub cim xeeb ua kom tsis muaj zog.
  5. Mob stroke Nyob rau hauv cov lus no tau to taub qhov kev ua txhaum tsis txaus ntawm lub hlwb mus ncig, uas yog qhov kev tuag ntawm qee thaj chaw.

Ntshav qab zib macroangiopathy ntawm qis qis entails xws li kev nthuav qhia:

  1. Mob hauv ceg.
  2. Qhov txhab mob txhab. Thaum lawv tshwm sim, kev ncaj ncees ntawm daim tawv nqaij yog qhov tsis taus.
  3. Tswm Yaj.
  4. Kev tuag ntawm cov ntaub mos muag. Thaum kev sib deev tshwm sim, txhais ceg yuav dub thiab poob nws lub luag haujlwm.

Cov hau kev kho

Lub hom phiaj ntawm kev kho cov kab mob no yog ua kom qeeb ntawm kev txhim kho kev phom sij los ntawm cov hlab ntsha, tuaj yeem ua rau muaj mob xiam oob qhab ntawm tus neeg mob lossis tuag. Lub hauv paus ntsiab lus tseem ceeb hauv kev kho tus mob no yog kev kho ntawm cov mob no:

  • Hypercoagulation;
  • Hyperglycemia;
  • Arterial kub siab;
  • Dyslipidemia.

Yuav kom cov roj metabolism hauv lub cev ua kom lwg li qub, cov neeg mob uas kuaj pom tau hais tias txoj kev kho mob insulin. Nws yuav tsum koom ua ke nrog tswj cov piam thaj hauv cov ntshav.

Txhawm rau txhawm rau txhim kho kev mob ntawm ib tus neeg, lipid-txo cov tshuaj yog kws kho. Cov no suav nrog fibrates, statins, tshuaj tiv thaiv kab mob. Ntawm tsis muaj qhov tseem ceeb me me yog kev pom ntawm kev noj zaub mov, uas cuam tshuam nrog kev txwv tsis pub cov tsiaj rog cov rog.

Nrog rau qhov kev hem thawj siab ntawm thromboembolic teebmeem, nws tsim nyog siv tshuaj tiv thaiv antiplatelet. Cov no suav nrog heparin thiab pentoxifylline. Cov kws kho mob feem ntau kho cov tshuaj acetylsalicylic acid.

Antihypertensive kev kho mob nrog kev kuaj mob no yog nqa tawm kom ua tiav thiab tswj kev ua haujlwm siab. Nws yuav tsum nyob tas li ntawm cov theem ntawm 130/85 hli RT. Kos duab. Yuav kom daws tau qhov teeb meem no, ACE inhibitors, captopril, yog siv.

Koj kuj yuav tsum tau siv tshuaj diuretics - furosemide, hydrochlorothiazide. Cov neeg mob uas tau myocardial infarction tau txais kev kho mob beta-blockers. Cov no suav nrog atenolol.

Kev kho qhov mob ntawm thiav txhab ntawm qhov mob kawg yuav tsum tau nqa tawm los ntawm kev saib xyuas ntawm ib tus kws phais neeg. Hauv kev mob vascular hnyav, kev saib xyuas hnyav yog muab. Yog tias muaj pov thawj, kev phais yuav ua tau.

Teeb meem

Qhov kev hem thawj ntawm macroangiopathy muaj tseeb dua hauv cov neeg muaj ntshav qab zib hom 2. Txoj kev pheej hmoo ntawm kev tuag los ntawm cov kab mob ntawm cov kab mob no yog 35-75%. Nyob rau hauv ib nrab ntawm cov mob, kev tuag tshwm sim los ntawm myocardial infarction.

Ntawm qhov phom sij txaus ntshai yog macroangiopathy ntawm cov hlab ntsha hlwb. Tus mob no ua rau mob ischemia mob siab.

Qhov tsis txaus ntseeg tau yog thaum 3 lub chaw vascular - lub hlwb, ceg, thiab lub siab - cuam tshuam ib txhij. Ntau tshaj ib nrab ntawm tag nrho cov haujlwm rau kev txiav txim siab ntawm cov qis qis yog cuam tshuam nrog macroangiopathy.

Nrog kev puas tsuaj ntawm ko taw, ulcerative tsis xws luag yog pom. Qhov no tsim ua ntej kev xav tau rau qhov tsim ntawm mob ntshav taw. Nrog kev puas tsuaj rau cov hlab ntsha cov hlab ntsha, cov hlab ntsha thiab cov pob txha pob txha, necrosis tau pom thiab cov txheej txheem purulent tshwm.

Cov tsos ntawm trophic mob rau sab hauv sab ceg yog vim muaj kev cuam tshuam ntawm cov hlab ntsha hauv cov hlab ntsha cuam tshuam ntawm ob txhais ceg. Qhov chaw khoob khoob khoob khawm feem ntau yog qhov chaw ntiv taw loj.

Kev hnov ​​mob nrog rau cov tsos mob ntawm tus mob ntshav qab zib tsis ua rau pom nws tus kheej ntau dhau. Tab sis thaum cov lus tim khawv tshwm, nws tsis tsim nyog rau ncua sijhawm ua haujlwm. Txawm tias qeeb me ntsis yog fraught nrog lub caij nyoog kho ntawm qhov txhab. Qee lub sijhawm nws yuav tsum tau ua ntau yam kev phais mob ntxiv.

Kev tiv thaiv kev ntsuas

Txhawm rau tiv thaiv cov tsos ntawm tus kab mob no, yuav tsum muaj cov lus pom zoo:

  1. Kho ntshav qab zib kom raws sijhawm;
  2. Ua raws li kev noj haus uas koom nrog kev txwv ntawm cov khoom noj protein, carbohydrates, ntsev thiab khoom noj muaj roj;
  3. Nquag lub cev hnyav;
  4. Tsis suav haus luam yeeb thiab haus cawv;
  5. Ua kom muaj kev tawm dag zog me ntsis, uas tsis yog qhov ua rau pom kev tshwm sim ntawm cov tsos mob ntawm angina pectoris;
  6. Txhua hnub, taug kev hauv huab cua ntshiab;
  7. Muab kev tshuaj ntsuam xyuas ntawm cov ntsiab lus lipid - ib zaug txhua 6 lub hlis;
  8. Ua qhov kev soj ntsuam zoo ntawm cov piam thaj hauv ntshav - qhov ntsuas no tau ntsuas ib hnub ib zaug.

Kev txhim kho ntawm macroangiopathy mob ntshav qab zib yog qhov xwm txheej tshwm sim. Cov kab mob no yog fraught nrog cov tsos ntawm phuaj txaus ntshai thiab tuaj yeem ua rau tuag taus. Yog li, nws yog ib qho tseem ceeb heev rau kev koom nrog nws kev tiv thaiv, thiab yog tias pom cov tsos mob tshwm sim, sab laj nrog tus kws kho mob tam sim ntawd.

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