Kev mob plawv cardiomyopathy: kev txhim kho thiab kev kho mob ntawm tus kab mob

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Qhov tsis zoo tshwm sim los ntawm ntshav qab zib ntawm cov kab mob plawv yog ncaj ncees rau kev nkag siab. Ntshav plawv mob plawv yog ib yam mob uas kho thiab kho tus mob myocardial kho kom zoo.

Vascular thiab lub plawv pathologies yog qhov tseem ceeb ua rau kev tuag thiab kev xiam oob qhab hauv 80% ntawm cov ntshav qab zib. Cardiomyopathy muaj qhov sib txuam nrog cov ntshav qab zib, nyob ntawm nws lub sijhawm.

Mob ntshav qab zib mellitus tsis cuam tshuam nrog lub hnub nyoog, mob ntshav nce ntshav thiab mob plawv.

Cov Ua rau Cardiomyopathy

Nyob rau hauv cov kev mob ntawm hyperglycemia, tsis muaj lub zog ntawm cov tsis muaj zog, yog li ntawd, kev ntxhov siab nyob rau hauv chav kawm ntawm oxidation thiab txo cov txheej txheem yog tsim. Cov kev tsis zoo no yog qhov ua kom mob ntshav qab zib cardiomyopathy.

Yog tias ib tus neeg muaj kab mob ntshav qab zib, tom qab ntawd muaj kev ua txhaum hauv lub siab, uas ua rau hnyav dua ntawm cov txheej txheem pathological ntawm myocardium.

Ntshav plawv mob plawv feem ntau yog tsim nyob rau hauv insulin-tiv thaiv cov neeg muaj ntshav qab zib mellitus thiab nquag heteroacidoses.

Xws li lub siab puas tsuaj yog qhov pom kev cuam tshuam los ntawm kev zom zaub mov hauv:

  • kev txo qis thiab oxidation kev cuam tshuam;
  • protein synthesis;
  • electrolyte metabolism;
  • muab cov hlwb nrog lub zog;
  • sib pauv ntawm kab kawm;
  • thauj cov pa oxygen hauv cov ntshav.

Ntshav plawv mob plawv yog tsim nyob rau hauv lub hauv paus ntawm ntau yam, uas tej zaum yuav:

  1. kev teeb tsa
  2. haumxeeb.

Hauv particular, nws provokes:

  • ua txhaum ntawm diastole thiab systole,
  • muaj nuj nqi mitochondrial,
  • tsawg dua kev cog lus ntawm cardiomyocytes.

Cov kev ua ntawm kev ua cov paib molecules uas ua lub luag hauj lwm rau lipid thiab piam thaj metabolism yuav ua rau tsawg.

Tomqab yug menyuam tawm mus kom tawv nqaij lub plawv tomqab yug menyuam tawm ntawm lub cev yug menyuam thaum yug menyuam. Txaus ntshai yog qhov siv tas li ntawm monodiet thiab tshaj qhov hnyav.

Kev tsim cov mob ntshav qab zib cardiomyopathy

Tus kab mob, raws li txoj cai, loj hlob qeeb thiab maj mam.

Lub siab tsis tuaj yeem muab kev so thiab tawm tsam, nws qhov tsis txaus.

Kev ua txhaum yog tshwm sim thaum lub sijhawm depolarization ntawm myocytes, nrog rau kev cuam tshuam hauv kev tsim khoom ntawm TSIS, uas tseem ua rau mob qhov xwm txheej.

Ntshav plawv mob plawv yog tshwm sim feem ntau yog los ntawm kev sib xyaw ua ke ntawm cov metabolic tsis txaus nrog vascular pathology.

Nws muaj ob hom pathology:

  1. thawj cov yam ntxwv cuam tshuam ntawm myocardial metabolic kev. Cov khoom uas muaj cov kab mob hauv qab-oxidized, cov roj ntsha tsis sib haum collagen, glucuronates, glycated protein ntau ntau ntxiv. Qhov no maj mam ua kom lub plawv tsis zoo thiab ua rau kev txhim kho tsis txaus nrog systolic lossis diastolic kawg,
  2. theem nrab tshwm vim yog mob ntshav qab zib angiopathy. Hauv kev kawm txog cov hlab ntsha, lawv cov sclerosis, thinning ntawm epithelium lossis kev nthuav tawm yog qhia tawm. Ib lub xeev ntawm cov pa oxygen tsis tu ncua ua rau cov kev ua haujlwm tsis haum uas cuam tshuam ncaj qha myocardial kev ua haujlwm.

Tam sim no, cov kws tshawb fawb tau ua pov thawj tias cov xwm txheej zoo li no ua rau lub luag haujlwm tsim kho pathology:

  • Lub caij nyoog hyperglycemia. Nrog nws, lub siab pib mob siab zuj zus. Feem ntau nws yuav siv sij hawm ntau xyoo rau tus mob kom pom nws tus kheej. Cov neeg mob feem ntau pom teeb meem vim yog cov tsos mob tas li.
  • Kev ua txhaum ntawm cov txheej txheem ntawm oxidation thiab rov qab sab hauv myocytes.
  • Kev cuam tshuam ntawm cov pa oxygen vim yog hloov pauv hauv tus qauv ntawm hemoglobin.

Cov piam thaj ntau hauv kev txiav ua rau lub siab ua rau lub plawv tsis muaj zaub mov tsis zoo. Cov txheej txheem ntawm lwm qhov tsim ntawm ATP nrog cov rog thiab cov protein pib.

Synthetic toxic metabolic khoom tau tsim tawm; lawv cuam tshuam tsis zoo rau kev ua haujlwm ntawm kev ua thiab cov leeg nqaij ntawm lub cev.

Symptomatology

Myocardial contractility tsawg dua li myocardial hlwb poob phaus.

Nyob rau lub sijhawm no, ib tus neeg yws yws ntawm kev mob hauv thaj av ntawm lub siab, uas dhau los ntawm lub sijhawm.

Tom qab qee lub sijhawm hauv cov ntshav qab zib, puffiness thiab ua tsis taus pa pib. Cov no yog cov tsos mob tseem ceeb qhia tias lub siab tsis ua haujlwm.

Mob hnyav rau ntshav qab zib yog:

  1. retinopathy
  2. nephroangiopathy.

Txij li thaum pib theem ntawm ntshav qab zib cardiomyopathy yog asymptomatic nyob rau hauv ntau yam, muaj lub sijhawm loj sib txawv nruab nrab ntawm cov tsos ntawm thawj qhov txawv txav ntawm tus mob tshwm sim thiab ua ntej cov txheej txheem kuaj mob.

Hauv cov neeg muaj hnub nyoog qis dua 40 xyoo, mob ntshav qab zib tuaj yeem kuaj pom nrog ECG ua kev tiv thaiv. Kwv yees li ntawm ib nrab ntawm cov xwm txheej ntawm txoj kev tshawb no qhia tau tias muaj kev ua txhaum hauv kev ua haujlwm ntawm lub siab.

Nws muaj cov qauv rau kev txiav txim siab cov ntshav qab zib mob plawv:

Kev txiav pauv ntawm cov hniav R thiab P,

  • cov kev hloov pauv ntawm T yoj vim thauj khoom,
  • cov kev hloov pauv hauv P-Q thiab Q-T ua ntu zus, QRS cov nyom, plawv dhia thiab conduction.

Kev mob tsis xwm yeem, tachycardia lossis bradycardia thiab lwm yam kev cuam tshuam yuav tshwm sim.

Kev kuaj mob thiab kho mob

Cardiomyopathy mob ntshav qab zib txhais tau hais tias koj yuav tsum nrhiav kom paub cov yam ntxwv ntawm tus kabmob.

Hauv qhov no, ib daim ntawv teev cov txheej txheem kuaj mob raug siv.

Kev qoj ib ce ua haujlwm tas li muaj qhov cuam tshuam zoo ntawm cov metabolism. Yog li, insulin tsis kam txo qis, thiab kev qab zib siab rau ntshav kuj tseem nce siab. Nrog rau kev ua lub neej nquag, koj tuaj yeem ua tiav hlawv cov roj ntsha dawb thiab siv cov ntshav qab zib.

Txhawm rau kom tus kws kho mob txhawm rau kuaj qhov tseeb, kev kuaj mob txuas ntxiv no yog qhov tsim nyog:

  1. kuaj mob thiab cov lus tsis txaus siab
  2. hluav taws xob hluav taws xob
  3. glucosuric thiab glycemic profile,
  4. txhua hnub saib xyuas ntawm ECG thiab ntshav siab,
  5. haumxeeb echocardiography,
  6. kev kawm txog lipid spectrum ntawm cov ntshav,
  7. Doppler echocardiography.

Kev kho mob rau cov ntshav qab zib mob plawv yog cov kev kho mob hauv qab no:

  • txhawm rau txhim kho cov kev cai ntawm kev kho tshuaj insulin, soj ntsuam cov qauv ntawm kev noj zaub mov noj ntshav qab zib mellitus thiab ua raws li kev ua haujlwm ntawm lub cev kom ua tiav qhov teeb meem glycemic,
  • kev siv tshuaj cardiotrophic thiab metabolic ntawm ATP, cov tshuaj potassium thiab L-carnitine npaj,
  • Cov vitamins B rau cov teebmeem neurotropic;
  • tshuaj uas txhim kho neuromuscular conduction.

Thaum muaj arrhythmia, antiarrhythmic tshuaj yuav tsum tau siv. Yog tias muaj cov cim ntawm lub plawv tsis ua haujlwm, thov:

  1. ACE inhibitor
  2. tshuaj kho mob
  3. mob glycosides.

Cov txheej txheem ntawm kev kho mob ntawm cov kab mob no tseem yog qhov nyuaj heev, vim tias koj yuav tsum nkag siab txog kev cuam tshuam ntawm cov khoom noj ntawm lub cev ntawm tib neeg lub cev. Txawm li cas los xij, nrog kev txheeb xyuas qhov tseeb ntawm qhov xwm txheej, nws muaj peev xwm ua tiav qhov ua tau zoo txaus siab thiab txhim kho lub neej kom zoo ntawm tus neeg mob.

Yog tias kev kho tshuaj tsis ua hauj lwm zoo, ces kev phais mob qee zaum siv tau rau kev kho mob nruj.

Nyob rau hauv kev kis mob hnyav tshaj plaws, tsuas yog txoj hauv kev muaj sia nyob yog kev hloov kho lub plawv.

Kev tiv thaiv kev ntsuas

Txog kev tiv thaiv, koj yuav tsum hloov koj txoj kev ua neej. Ib tus neeg mob yuav tsum pib qoj ib ce kom nruj thiab soj qab xyuas lawv cov zaub mov kom yuag.

Tus neeg mob ntshav qab zib yuav tsum tau ntsuas kom tshem tawm hyperglycemia, nrog rau kev tshem tawm:

  • fatty acid cov ntsiab lus
  • insulin kuj.

Tam sim no, cov kws tshawb fawb tsis muaj kev teeb tsa cov kev paub dav dav, txij li qhov tsim nyog ntawm kev tshawb fawb txog kev hloov pauv ntawm lub xeev ntawm cov ntshav qab zib mob plawv thaum pib tsis tau ua.

Yog tias muaj lub siab nyiam lub plawv tsis ua haujlwm, nws yog ib qho tseem ceeb kom tso tseg txoj kev haus cawv, kev nqus ntawm qhov uas ua rau lub nra hnyav ntxiv. Nws yog ib qho tsim nyog los tiv thaiv lub cev, kom tsis txhob muaj lub cev ntau dhau thiab tsis muaj kev kub ntxhov.

Koj tuaj yeem sab laj nrog tus kws qhia zaub mov noj kom paub txog kev noj haus ntawm cov tib neeg. Nws kuj tseem ceeb kom hloov kho txoj kev ua neej thiab txiav kom tsis txhob haus luam yeeb thiab haus dej haus cawv. Koj yuav tsum pib qoj ib ce kho mob ntshav qab zib mellitus, piv txwv li, taug kev lossis ua luam dej.

Txhawm rau tshem tawm qhov tsis zoo uas cuam tshuam rau lub siab, cov tshuaj ib txwm siv tau. Kev ua kom mob siab pab txo qis thiab tshem tawm lub plawv kev ntxhov siab.

Nws yog qhov tseem ceeb rau haus tincture nrog viburnum thiab zib ntab rau txog 30 hnub. Cov dej haus haum haum rau tag nrho cov txheej txheem hauv lub cev. Kev siv lwm txoj kev tiv thaiv kev tiv thaiv yuav tsum pom zoo nrog tus kws kho mob.

Daim vis dis aus hauv tsab xov xwm no hais txog cov tsos mob, ua rau, thiab kho mob plawv.

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