Cov ntshav qab zib tom qab noj mov

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Glucose yog ib qho tseem ceeb monosaccharide uas ib txwm muaj nyob hauv tib neeg lub cev thiab, koom nrog ntau ntawm cov txheej txheem biochemical, npog lub zog noj ntawm cov hlwb thiab cov nqaij. Cov piam thaj nkag nrog cov zaub mov lossis tau tsim los siv glycogen tso rau hauv lub siab thiab qee yam khoom hauv nruab nrog cev.

Tus nqi Glycemia yuav txawv txhua hnub. Lawv nyob ntawm tus neeg lub hnub nyoog, nws cov kab ke thiab lub cev qhov hnyav, lub sijhawm noj mov dhau los, muaj cov kab mob pathological, ua si lub cev. Tom ntej no, dab tsi yog cov txiaj ntsig ntawm cov piam thaj hauv ntshav tom qab noj mov, lub cev muaj sia thiab pathological ua rau nws nce ntxiv, nrog rau cov txheej txheem kho.

Vim li cas lub cev xav tau cov kua nplaum?

Cov piam thaj (qab zib) yog ib cov carbohydrate yooj yim uas tau txais thaum lub sijhawm tawg ntawm polysaccharides. Hauv plab hnyuv me, nws nqus tau rau hauv cov ntshav, tom qab ntawd nws kis tau los ntawm lub cev. Tom qab cov piam thaj nyob hauv cov ntshav tom qab noj mov hloov mus rau hauv sab nraud, lub paj hlwb xa ib lub teeb liab rau cov kab mob ntshav siab (insulin) yuav tsum tau tso tawm rau hauv cov ntshav.

Cov tshuaj insulin yog yam tshuaj uas lub cev ua haujlwm yog lub zog tswj cov kev faib tawm saccharide hauv lub cev. Nrog nws cov kev pab, cov tubules tshwj xeeb qhib rau hauv cov hlwb uas cov piam thaj hla hauv. Nyob ntawd nws tawg rau hauv dej thiab lub zog.


Insulin - qhov tshwj xeeb "qhov tseem ceeb" rau lub monosaccharide

Tom qab cov ntshav piam thaj tau poob qis, ib lub teeb liab tau txais hais txog qhov yuav tsum tau rov qab los rau nws qib siab. Cov txheej txheem ntawm cov kua nplaum ua ke pib, uas lipids thiab glycogen koom nrog. Yog li, lub cev tab tom sim rov qab glycemia rau dab tsi.

Tseem Ceeb! Cov neeg tseem ceeb ntawm cov piam thaj yog lub paj hlwb cov hlwb. Yog hais tias nws qhov ntau yog qhov tsis txaus, lub zog tshaib plab tshwm sim, uas ua rau pom qhov tshwm sim ntawm pathological.

Kev noj ntshav qab zib ntau dhau kuj tsis zoo. Hauv cov khoom loj, cov monosaccharide muaj peev xwm ua kom muaj cov tshuaj lom, txij li tiv thaiv keeb kwm ntawm hyperglycemia, cov txheej txheem ntawm cov kua nplaum nyob nrog lub cev cov protein ua haujlwm. Qhov no hloov pauv lawv tus cwj pwm ntawm lub cev thiab lub cev, ua rau qeeb qeeb rov qab.

Qhov ntsuas hloov pauv li cas thoob plaws ib hnub

Cov piam thaj hauv ntshav tom qab noj mov, ntawm lub plab khoob, tom qab kev qoj ib ce hloov nws tus lej. Thaum sawv ntxov, yog tias cov zaub mov tseem tsis tau nkag mus rau hauv lub cev, cov cim hauv qab no (hauv mmol / l):

  • qhov tsawg kawg nkaus uas tso cai rau cov poj niam laus thiab txiv neej yog 3.3;
  • tso cai ntau kawg nkaus hauv cov neeg laus yog 5.5.

Cov duab no ib txwm muaj rau hnub nyoog txij 6 txog 50 xyoo. Rau cov menyuam mos thiab cov menyuam mos, qhov ntsuas tsis sib txawv - ntawm 2.78 txog 4.4. Rau cov menyuam muaj hnub nyoog pib kawm ntawv, qhov siab tshaj yog 5, qhov ntsuas qis qis yog zoo ib yam li cov hnub nyoog nruab nrab.

Tom qab 50 xyoo, ntsuas hloov me ntsis. Nrog lub hnub nyoog, qhov kev txwv txiav txim siab nce mus, thiab qhov no tshwm sim nrog txhua kaum xyoo tom ntej. Piv txwv, cov ntshav qabzib hauv cov neeg laus dua 70 yog 3.6-6.9. Qhov no yog txiav txim siab zoo tus xov tooj.


Txhua tus neeg hauv tsev neeg muaj glycemia ntsuas uas zoo rau nws hnub nyoog.

Cov ntshav qab zib los ntawm cov leeg ntshav yog siab dua me ntsis (kwv yees li 7-10%). Koj tuaj yeem tshawb xyuas qhov ntsuas tsuas yog nyob hauv chav kuaj. Cov txheej txheem (hauv mmol / l) yog cov lej txog 6.1.

Lub sijhawm sib txawv

Ib qho ntawm cov kabmob uas nyiam tshaj plaws uas ua rau nws tus kheej muaj suab thaj ntau yog ntshav qab zib. Txhua tus neeg mob ntshav qab zib paub tias glycemia yuav tsum tau tswj hwm ntawm cov sij hawm sib txawv thoob plaws ib hnub. Qhov no yuav tso cai rau koj xaiv cov tshuaj kom yog, los tiv thaiv kev ua kom tsis meej.

Qhov 1 ntawm tus kab mob yog tus cwj pwm los ntawm qhov tseeb tias hyperglycemia tshwm sim vim kev ua haujlwm tsis txaus ntawm cov tshuaj insulin. Hom 2 tshwm sim vim yog qhov tshwm sim ntawm insulin tsis kam (poob ntawm cov tshuaj hormones tsis txaus rau lub cev lub cev). Pathology tuaj yeem nrog cov qib nrawm hauv qab zib thoob plaws ib hnub, yog li nws yog qhov tseem ceeb uas yuav tsum paub cov cai tso cai (hauv mmol / l):

  • tom qab so hmo hauv cov laus - txog 5.5, hauv cov menyuam yaus hnub nyoog qis dua 5 xyoos - txog 5;
  • ua ntej cov zaub mov nkag mus rau hauv lub cev - nce txog 6, hauv cov menyuam yaus - txog 5.5;
  • tam sim ntawd tom qab noj mov - nce txog 6,2, menyuam yaus lub cev - nce txog 5,7;
  • hauv ib teev - txog 8.8, hauv ib tus menyuam - txog 8;
  • tom qab 120 feeb - nce txog 6.8, hauv cov menyuam - nce txog 6.1;
  • ua ntej hmo so - nce txog 6.5, hauv tus menyuam - nce txog 5,4;
  • thaum tsaus ntuj - txog 5, cov me nyuam lub cev - nce txog 4.6.
Tseem Ceeb! Cov piam thaj ntau npaum li cas nyob hauv cov zis yog lwm qhov tseem ceeb rau kev kuaj xyuas, uas tau hais meej raws li cov ntshav qabzib nyeem. Hauv cov menyuam muaj hnub nyoog noj qab haus huv thiab tus neeg loj, qib no yuav tsum sib npaug 0, thaum cev xeeb tub mus txog 1.6 yog tso cai.

Kawm paub ntau ntxiv txog cov ntshav qab zib cov txiaj ntsig thaum lub cev xeeb tub los ntawm cov kab lus no.

Cov ntshav qabzib tom qab noj mov

Tom qab noj ntshav qab zib, cov neeg hauv qab no yuav tsum tau saib xyuas:

  • nyob ib puag ncig pathological lub cev hnyav;
  • muaj tus neeg mob ntshav qab zib los ntawm kab keeb kwm;
  • muaj tus cwj pwm tsis zoo (haus cawv, haus luam yeeb);
  • cov neeg uas nyiam kib, haus zaub mov, zaub mov nrawm;
  • kev txom nyem los ntawm arterial tawg thiab muaj roj cholesterol siab;
  • cov poj niam uas tau yug menyuam cov menyuam hnyav dua 4 kg ua ntej.

Qabzib ntxiv nyob rau hauv cov ntshav tom qab kev noj haus yog qhov ib txwm ua rau lub cev noj qab nyob zoo

Yog tias glycemia pauv nce ntau zaus, koj yuav tsum nrhiav kev tawm tswv yim ntawm tus kws kho endocrinologist. Nws yog ib qho tsim nyog kom nrog tus kws kho mob tham, ua cov kev tshawb fawb ntxiv yog tias muaj lub zog pathological rau haus, noj. Tib lub sijhawm, ib tug neeg feem ntau tso zis thiab tsis nce phaus lub cev, ntawm qhov tsis tooj, qhov txo qis hauv lub cev hnyav yog ua tau.

Tsis tas li ntawv ceeb toom yuav tsum yog qhov zoo nkaus ntawm lub cev qhuav thiab nruj ntawm daim tawv, qhov tsos ntawm cov kab nrib pleb ntawm cov ces kaum ntawm daim di ncauj, qhov mob ntawm qhov qis qis, tshwm sim ntawm cov tawv nqaij tsis meej uas tsis zoo rau lub sijhawm ntev.

Tseem Ceeb! Cov tsos mob saum toj no qhia hyperglycemia thiab tej zaum yuav yog tsos mob ntshav qab zib.

Ib qho txiaj ntsig tsis txaus ntawm cov piam thaj nyob rau sab nraud tus txheej txheem yuav qhia tau qhov kev txhim kho ntawm cov tshuaj tiv thaiv insulin, uas tseem tau kuaj xyuas los ntawm kev tshawb fawb txog kev ntsuas (qab zib load kuaj). Tus mob no yog hu ua mob ntshav qab zib. Nws yog tus cwj pwm los ntawm qhov tsis xws luag rau qhov tshwm sim ntawm ib hom tshuaj insulin-ywj pheej ntawm "mob qab zib".

Vim li cas thiaj tuaj yeem muaj cov qab zib tom qab noj mov tag?

Txhua tus tau siv rau qhov tseeb tias khoom noj khoom haus ua rau muaj kev nce siab ntxiv hauv cov piam thaj, tab sis kuj tseem "rov qab sab ntawm npib." Qhov no thiaj li yog tshuaj tiv thaiv kab mob ntshav qab zib (hypoglycemia). Feem ntau, nws tshwm sim tawm tsam keeb kwm ntawm kev rog lossis nrog hom 2 mob ntshav qab zib mellitus.


Kev tawm hws yog ib qho ntawm cov tsos mob ntawm tus mob hypoglycemia.

Cov kws tshawb fawb tsis tuaj yeem nyob ntawm qhov laj thawj ntawm qhov xwm txheej no, yog li lawv tau txheeb xyuas ntau qhov theories ntawm nws txoj kev txhim kho:

Yuav kuaj kab mob ntshav qab zib li cas
  1. Kev noj haus uas ib tus tib neeg ua rau cov khoom noj tsis haum rau lub cev kom yuag poob. Yog tias lub cev tsis tau txais "lub tsev tsim" cov qauv hauv polysaccharides rau lub sijhawm ntev, nws pib siv nws cov peev txheej, npaj cia tshwj tseg. Tab sis lub caij dhau los thaum lub tsev khaws khoom lag luam tau khoob, vim nws tsis tau ntxiv rov qab.
  2. Pathology, nrog los ntawm intolerance rau fructose ntawm ib tug muaj keeb xwm.
  3. Nws feem ntau tshwm sim hauv cov neeg uas tau phais plab hnyuv yav dhau los.
  4. Tawm tsam keeb kwm yav dhau los ntawm cov teeb meem ntxhov siab, spasm ntawm tus txiav mob tshwm sim, uas ua rau kev sib txuas ntawm cov kua dej hauv cov khoom loj.
  5. Qhov muaj cov tshuaj insulinomas yog qhov hlav ntawm cov qog ntshav uas tswj tsis tau cov tshuaj insulin rau hauv cov hlab ntshav.
  6. Qhov ua kom tsawg ntawm cov piam thaj hauv glucagon, uas yog insulin antagonist.

Reactive hypoglycemia nthuav dav. Ib tug neeg sau ntawv tshwm sim ntawm insomnia, kiv taub hau, tawm hws ntau dhau. Nws pheej xav noj, txawm tias tom qab nws noj su, noj hmo. Kev yws yws ntawm kev qaug zog, txo qis kev ua haujlwm.

Txhawm rau tshem tawm cov mob no, koj yuav tsum hloov koj txoj kev ua neej: noj ntau, tab sis nyob rau qee qhov me me, tsis kam lees sai cov roj carbohydrates, soj ntsuam lub ntsiab cai ntawm khoom noj khoom haus, uas cov kua dej tso tawm hauv qhov txaus. Nws yog ib qho tsim nyog kom tso tseg dej cawv thiab kas fes.

Nws yog ib qho tseem ceeb ntawm kev ua kis las, tab sis tsis txhob tsim txom lub nra. Yuav kom nce qab zib, glucagon tau txhaj tshuaj.

Zais plab tsis zoo tom qab noj mov

Tus mob no yog hu ua postprandial hyperglycemia. Nws yog tus yam ntxwv ntawm cov piam thaj hauv cov ntshav tom qab noj mov saum toj 10 mmol / L. Cov ntsiab lus hauv qab no yog suav hais tias yog pheej hmoo txaus ntshai:

  • phaus pathological;
  • ntshav siab;
  • cov naj npawb siab ntawm cov tshuaj insulin hauv cov ntshav;
  • muaj cov "cholesterol" tsis zoo;
  • tsis hnov ​​lus qab zib kom tsis taus;
  • predisposition ntawm keeb muaj keeb;
  • yog txivneej lossis pojniam (feem ntau tshwm sim hauv txivneej).

Siab glycemia ob peb teev tom qab noj mov - cov pov thawj ntawm cov txheej txheem pathological hauv lub cev
Tseem Ceeb! Cov kev tshawb fawb soj ntsuam tau lees tias qhov tseem ceeb ntawm qhov tsis muaj tom qab tau txais qhov tshwm sim hyperglycemia kom ua tiav cov nyiaj them rov qab thiab qhia meej tias qhov no pom tau tias tseem ceeb tshaj qib ib txwm ntawm glycated hemoglobin.

Txog tav su hyperglycemia yog txuam nrog kev pheej hmoo ntawm kev tsim cov mob hauv qab no:

  • macroangiopathies - kev puas tsuaj rau cov hlab ntsha loj;
  • retinopathy - pathology ntawm cov hlab ntsha ntawm pob nyiaj;
  • kev nce hauv thickness ntawm cov carotid cov hlab ntsha;
  • oxidative kev nyuaj siab, o, thiab endothelial kawg;
  • txo hauv kev ntshav txaus hauv cov leeg plawv;
  • oncological kev ntawm malignant xwm;
  • pathology ntawm cov kev paub txog kev ua haujlwm hauv cov neeg laus lossis ntawm keeb kwm ntawm ib hom tshuaj insulin-ywj pheej.

Tseem Ceeb! Postprandial hyperglycemia coj teeb meem loj rau tib neeg kev noj qab haus huv, xav kom muaj kev kho kom zoo.

Kev sib ntaus ntawm cov kab mob pathology muaj nyob rau hauv kev noj zaub mov noj nrog cov muaj cov carbohydrate tsawg, nyob rau hauv kev sib ntaus cov tiv thaiv lub cev muaj ceeb thawj, hauv kev siv khoom siv thauj khoom. Cov tshuaj uas pab ua kom tshem tawm cov ntshav qab zib tom qab noj:

  • amylin analogs;
  • DPP-4 inhibitors;
  • av nplaum;
  • derivatives ntawm glucagon-zoo li peptide-1;
  • insulins.

Kev siv tshuaj yeeb yog ib qho ntawm cov txheej txheem los pab tus neeg mob txhawm rau nce ntshav hauv qab zib

Cov thev naus laus zis tso cai rau koj los tswj cov glycemia tsis yog hauv chav kuaj xwb, tab sis kuj nyob hauv tsev. Txhawm rau ua qhov no, siv glucometers - cov khoom siv tshwj xeeb, uas suav nrog cov ntawv me me rau cov ntiv tes hno thiab ntsuas cov khoom siv coj los ua cov tshuaj lom biochemical thiab ntsuas cov suab thaj.

Txhawb txoj qib qub ntawm glycemia hauv cov hlab ntsha, tsis yog ua ntej xwb, tab sis kuj tom qab noj mov, suav hais tias yog qhov tseem ceeb rau kev tiv thaiv kev txhim kho ntawm cov teeb meem ntawm ntau cov kab mob pathological.

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