Cov neeg uas muaj tus mob ntshav qab zib hom 2 tsis yog ib txwm muaj peev xwm tswj cov ntshav qabzib hauv lub cev los ntawm kev noj haus thiab kev tawm dag zog. Yog li, qee tus neeg mob ntshav qab zib yuav tsum tau siv cov tshuaj tiv thaiv ntshav qab zib hauv lub cev, tshwj xeeb Glidiab.
Cov tshuaj no pab ua kom lub cev nyob hauv cov ntshav khov zoo, thiab nce ntxiv ntawm cov khoom noj kom muaj txiaj ntsig yog tias cov ntshav qab zib ua mob yuag dhau. Tsis tas li ntawd, thaum siv Glidiab MV 30, lub siab ntawm cov kua dej tso kua mis rov qab.
Kuv tuaj yeem yuav cov tshuaj Glidiab ntau npaum li cas? Hauv cov tsev muag tshuaj, tus nqi ntawm cov tshuaj yog 120-200 rubles. Tus nqi yuav nce raws tus nqi ntawm cov khoom xyaw nquag hauv cov ntsiav tshuaj. Glidiab MB 30 mg thiab 80 mg yog lag luam muag.
Tus txheej txheem ntawm kev nqis tes ua thiab kev taw qhia rau kev siv
Glidiab MV yog ib tus neeg sawv cev hypoglycemic rau cov pab pawg ntawm sulfonylurea derivatives ntawm 2 tiam. Cov tshuaj muaj gliclazide thiab excipients. Glyclazide hauv ib ntsiav tshuaj muaj 80 mg lossis 30 mg.
Cov tshuaj tiv thaiv tsis ua hauj lwm li cas? Glyclazide raws li qhov nqus tau stimulates cov leeg glycogen synthetase kev ua si thiab cov tshuaj insulin zais. Tsis tas li, cov tshuaj no potentiates insulin secretory nyhuv ntawm cov piam thaj, thiab ua rau muaj kev nce siab hauv qhov rhiab ntawm cov ntaub so ntswg mus rau insulin.
Ntxiv mus, gliclazide pab txo cov kis ntawm kev noj zaub mov thiab pib ntawm lub cev ua haujlwm ntawm cov kua dej insulin. Yog tias koj saib cov lus qhia rau kev siv rau Glidiab, koj tuaj yeem pom tias thaum koj siv cov ntsiav tshuaj, lub siab ntawm hyperglycemia poob qis, thiab thaum ntxov ntawm cov tshuaj insulin tso rov qab.
Tag nrho cov yam cuam tshuam ncaj qha cuam tshuam rau cov metabolism hauv carbohydrate thiab microcirculation. Yog tias koj ntseeg tias cov lus qhia, tom qab ntawd Glidiab MV pab txo qis rau kev txhawm thiab txhawm rau platelets, thiab tib lub sijhawm ua rau vascular permeability. Yooj yim muab, nrog kev siv cov ntsiav tshuaj, qhov ntxim nyiam ntawm kev kho mob ntawm microthrombosis thiab atherosclerosis yog txo qis.
Cov kev tshawb fawb kuj tau pom tias tus neeg sawv cev hypoglycemic pab ua kom qeeb ntawm kev txhim kho ntawm cov teeb meem xws li mob ntshav qab zib ntawm lub cev tsis nyob ntawm theem. Ntxiv mus, siv Glidiab MV cov ntsiav tshuaj tuaj yeem cuam tshuam rau kev ua haujlwm ntawm kev noj zaub mov noj rau cov neeg mob kev rog dhau los.
Kuv xav nco ntsoov tias cov tshuaj metabolites ntawm cov tshuaj tau nthuav tawm ua ke nrog cov zis hauv ib daim ntawv hloov pauv, thiab ua ke nrog quav hauv daim ntawv ntawm metabolites.
Cov lus qhia rau kev siv tshuaj
Nyob rau hauv rooj plaub dab tsi yog nws raug nquahu kom siv Glidiab 80 ntsiav tshuaj? Cov lus qhia tau hais tias nws raug nquahu kom siv cov tshuaj hauv kev kho mob hom 2 ntshav qab zib mellitus, yog tias kev noj zaub mov noj thiab kev tawm dag zog tsis pab tswj cov ntshav qabzib.
Ua nrog rau lwm cov tshuaj, Glidiab MB tsis tshua muaj neeg siv. Tab sis nws yuav tsum nco ntsoov tias qhov ua rau lub ntsej muag hypoglycemic ntawm cov tshuaj tuaj yeem nce ntxiv yog tias, nrog rau kev siv tshuaj kho mob, kev noj haus thiab kev ua si kis las yog qhov sib npaug.
Yuav noj li cas? Qhov hno koob yog 80 mg. Ntxiv mus, qhov ntau zaus ntawm kev siv yog 2 zaug hauv ib hnub - thaum sawv ntxov thiab yav tsaus ntuj. Nws raug nquahu kom noj cov ntsiav tshuaj 30-60 feeb ua ntej noj mov.
Yog tias qhov koob tshuaj tsawg kawg nkaus ntawm 80 mg tsis muaj ib qho kev tiv thaiv hypoglycemic, ces qhov muab tshuaj yog maj mam nce. Feem ntau, rau hom ntshav qab zib hom 2, koob tshuaj 160 mg yog qhov zoo tshaj. Qhov siab tshaj plaws tau ntau npaum li cas ntawm cov tshuaj yog 320 mg.
Tab sis peb yuav tsum nco ntsoov tias nrog rau cov koob tshuaj ntau ntxiv, qhov ntxim nyiam ntawm kev muaj ntshav qab zib thiab lwm yam mob hnyav nce ntxiv.
Kev sib cuam tshuam txog yeeb tshuaj thiab Tshuaj Tiv Thaiv
Nws yuav tsum nco ntsoov hais tias ib tug xov tooj ntawm cov tshuaj muaj peev xwm nce hypoglycemic nyhuv ntawm yeeb tshuaj Glidiab MB. Yog li, cov lus qhia tau sau tseg tias cov tshuaj yuav tsum yog ua tib zoo ua ke nrog histamine H2-receptor blockers, tshuaj tsis-steroidal anti-inflammatory tshuaj, antifungal agents, ACE inhibitors.
Cov tshuaj tiv thaiv tuberculosis, beta-adrenoblockers, nphav phav phav hom anticoagulants, anabolic steroids, MAO inhibitors, salicylates thiab lwm tus kuj tseem tuaj yeem ua rau muaj kev tiv thaiv hypoglycemic.
Tias yog vim li cas ua ntej siv cov ntsiav tshuaj Glidiab, koj yuav tsum ua tib zoo kawm cov lus qhia thiab sab laj tus kws kho mob.
Ntawm cov contraindications kom siv cov ntsiav tshuaj yuav qhia tau:
- Ntshav Qab Zib Hom 1 yog insulin-vam khom.
- Ntshav qab zib ketoacidosis.
- Xav sim lossis tsis nco qab. Ntxiv mus, ib qho nruj contraindication yog hyperosmolar coma.
- Leukopenia
- Lub sijhawm cev xeeb tub.
- Lub sijhawm lactation.
- Mob rau hepatic thiab lub raum tsis ua haujlwm.
- Cov xwm txheej uas nrog ua ke los ntawm kev ua txhaum ntawm cov txheej txheem ntawm kev nqus ntawm cov khoom noj, thiab kev txhim kho ntawm lub qog ntshav qab zib. Cov mob no suav nrog kev cuam tshuam ntawm cov hnyuv, paresis ntawm lub plab thiab cov kab mob kis.
- Ua xua rau cov khoom xyaw ntawm cov ntsiav tshuaj.
- Tej yam mob uas yuav tsum tau siv tshuaj insulin. Cov mob no suav nrog hlawv, raug mob, lossis phais mob.
- Kev quav cawv
- Lub siab ntsws Febrile.
Tsis tas li, cov tshuaj yuav tsum tau siv nrog ceev faj hauv cov neeg kev txom nyem los ntawm cov thyroid tsis ua hauj lwm.
Kev txheeb xyuas thiab cov kev mob tshwm sim ntawm Glidiab
Qhov kev ntsuam xyuas txog Glidiab yog dab tsi? Tus mob ntshav qab zib tau teb zoo rau cov tshuaj. Coob tus neeg tau txais kev txaus siab los ntawm cov nqi pheej yig ntawm cov tshuaj, thiab cov ntsuas kev ua tau zoo.
Ntxiv mus, Glidiab, raws li cov neeg mob ntshav qab zib, yog qhov zoo vim tias nws tsis tshua muaj kev phiv los ntawm kev siv tshuaj tsawg. Lwm qhov kev ntaus nqi ntawm cov tshuaj rau cov tib neeg yog qhov tseeb tias nws nce lub txiaj ntsig ntawm kev kho kev noj haus.
Cov kev phiv dab tsi tuaj yeem ua rau cov tshuaj hypoglycemic ua rau? Raws li cov lus qhia, cov tshuaj tuaj yeem ua rau:
- Kev ua txhaum ntawm endocrine system. Lawv qhia raws li kev mob ntshav qab zib. Tab sis nws yuav tsum raug sau tseg tias qhov kev lees paub no tsuas yog tshwm sim nrog kev xaiv tsis zoo ntawm qhov tshuaj.
- Kev tsis haum xeeb, nkees nkees, tawm tsam kev tawm tsam, ua kom lub cev ntaj ntsug, mob taub hau, kiv taub hau, nce zog.
- Tsawg dua pom pom tseeb tseeb.
- Cov Niampaus.
- Lub Neej Bradycardia
- Ua pa.
- Ncuav Qab Zib.
- Thrombocytopenia, ua haujlwm tsis txaus, leukopenia.
- Kev ua xua tsis haum.
- Digestive system malfunction. Ib tus neeg yuav mob raws plab, qhov kev xav nyob rau hauv thaj chaw epigastric, xeev siab, tsis nco qab, mob cholestatic daj ntseg, nce kev ua haujlwm ntawm hepatic transaminases.
Feem ntau, cov kev mob tshwm sim los daws lawv tus kheej tom qab tsum tsis haus yeeb tshuaj thiab tau txais kev kho mob zoo tsim nyog.
Qhov zoo tshaj plaws analogue ntawm Glidiab
Cov analogues ntawm Glidiab yog dab tsi? Hloov chaw, ntau hom kab mob hypoglycemic raws li metformin hydrochloride tuaj yeem siv. Pab pawg neeg tau txais txiaj ntsig zoo heev yog Formine. Cov tshuaj no yog Glidiab's hloov zoo tshaj plaws.
Tus nqi ntawm cov tshuaj yog kwv yees li 180-260 rubles. Formmetin muaj nyob rau hauv qhov ntau npaum ntawm 500 mg, 850 mg thiab 1 gram. Ib pob muaj 60 ntsiav tshuaj. Cov tshuaj muaj pes tsawg leeg suav nrog metformin hydrochloride, povidone, primellose, magnesium stearate.
Qhov nquag ua haujlwm ntawm formin ua li cas? Cov lus qhia tau qhia tias metformin hydrochloride inhibits cov txheej txheem ntawm gluconeogenesis nyob rau hauv daim siab, thiab pab txo qis kev nqus ntawm cov piam thaj los ntawm cov hnyuv.
Tsis tas li, cov nquag tiv thaiv txhim kho kev siv hluav taws xob ntau ntawm cov piam thaj, thiab pab txhawm rau nce cov rhiab ntawm cov ntaub so ntswg mus rau qhov cuam tshuam ntawm insulin. Hauv qhov no, metformin hydrochloride tsis cuam tshuam rau cov txheej txheem ntawm insulin secretion los ntawm beta hlwb, vim tias qhov ntxim nyiam ntawm kev txhim kho hypoglycemia txo qis.
Nrog kev pab ntawm Formetin, hom 2 mob ntshav qab zib tuaj yeem kho tau. Tshwj xeeb feem ntau, cov tshuaj yog siv thaum muaj tus mob ntshav qab zib ua mob rog thiab nyob rau kis uas kev noj zaub mov tsis pab ua kom cov ntshav qabzib tseem ceeb. Kuv xav nco ntsoov tias cov tshuaj feem ntau siv ua ke nrog cov tshuaj cuam tshuam nrog sulfonylurea derivatives.
Yuav noj li cas? Thawj koob tshuaj yog 1000-1700 mg ib hnub twg. Ntxiv mus, kev muab tshuaj tau muab faib ua 2 koob. Nws yog qhov zoo dua rau kev noj tshuaj tom qab noj mov nrog qhov dej ntau.
Yog tias cov piam thaj hauv ntshav tsis ruaj, ces cov tshuaj siv tshuaj maj mam nce mus rau 2-3 grams hauv ib hnub. Qhov siab tshaj uas tau tso cai niaj hnub rau Formetin yog 3 grams, tsis ntau dua. Tab sis cov neeg mob laus yog pom zoo kom haus tsis ntau tshaj 1 gram nyob rau ib hnub.
Cov kev txwv tsis pub siv rau qhov tshuaj:
- Kev fab tshuaj rau cov Cheebtsam.
- Lub raum khiav tsis zoo, hauv lub raum tsis ua haujlwm.
- Cov theem mob myocardial infarction.
- Lub cev qhuav dej.
- Plawv lossis ua pa tsis ua haujlwm.
- Cerebrovascular kev huam yuaj.
- Kev haus cawv mus ntev
- Cev xeeb tub thiab lactation.
- Cov mob uas muaj qhov xav tau ntawm kev siv tshuaj insulin. Nws tuaj yeem yog cov kev raug mob hnyav, hlawv lossis phais mob.
- Lactic acidosis.
- Ua raws li kev noj zaub mov nruj, uas muab rau kev txo qis hauv calories txhua hnub mus txog 1000 kilocalories.
- Daim ntawv thov lub sijhawm 2 hnub dhau los ntawm kev tshawb nrhiav xoo hluav taws xob nrog kev qhia ntawm kev sib piv ntawm iodine muaj cov tshuaj muaj yees. Los ntawm txoj kev, cov tshuaj yuav tsum tsis txhob noj 2 hnub ua ntej xws li kev xoo hluav taws xob.
Ntawm cov kev mob tshwm sim ntawm cov tshuaj, muaj kev tsis sib haum xeeb hauv kev ua haujlwm ntawm lub plab zom mov, kev mob plab zom mov, ua rau lub cev tsis muaj ntshav, tsis muaj ntshav qab zib thiab kev fab tshuaj tsis haum. Daim vis dis aus hauv tsab xov xwm no yuav qhia koj tias tshuaj dab tsi muaj ntshav qab zib.
